The Tricaprin Research Blog

Millions of people taking GLP-1 drugs notice surprising changes in how their face looks. Here is the science behind why it happens, what it means for your health, and what you can do about it.

Most dietary fats take hours to become useful energy. C10 (Tricaprin) takes a different route entirely — entering your cells' power plants directly and triggering a cascade of metabolic effects no other fat can match.

Your body already makes the world's most effective energy-regulating hormone. C10 Tricaprin tells it when to release — triggering a cascade of clean, sustained fuel that drug-based GLP-1 therapies try to replicate artificially.

The MCT oil on your shelf is mostly C8. But a landmark 2023 study found that C8 cannot enter skeletal muscle mitochondria without carnitine — the very bottleneck that aging creates. Here's what the science actually says.

Most strength research was done on young adults. A decade-long Japanese clinical trial series tested frail adults averaging age 85 on just 6g of C8/C10 MCTs per day — and the results were measurable and significant.

In 1966, a Norwegian trial found that tricaprin reduced atherosclerotic plaque. In 2025, a Nature Cardiovascular Research registry documented 100% three-year survival in heart failure patients given the same compound.

GLP-1 receptor agonists produce dramatic weight loss — but up to 40% of that lost weight is muscle, not fat. The mechanism is predictable and well-documented. So is the solution.

GLP-1 medications can cause significant muscle loss alongside fat loss. Science shows that protein and resistance training can protect lean mass — here is what you need to know.

Do GLP-1 drugs slow your metabolism? The answer is nuanced — these drugs don't directly reduce calorie burn, but weight loss always does. Here is what the research actually shows.